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News article by Dr Abhishek Katakwar published in Deccan Chronicle (Date 21 january 2018)
Our complete gastrointestinal tract is lined with microbes collectively called the microbiome, which includes bacteria, fungi, and even viruses. Though it sounds weird and even unhealthy, gut bacteria perform many important functions in the body, including aiding the immune system, producing the feel-good brain chemical serotonin, making energy available to the body from the food we eat, and disposing of foreign substances and toxins, though we always have a mixture of good and bad bacteria, sometimes the bad guys get the upper hand, causing an imbalance in gut bacteria, and can cause severe health problems than just stomach pain, gas, bloating, or diarrhea. Because 60-80% of our immune system is located in our gut, gut imbalances have been linked to obesity, hormonal imbalances, autoimmune diseases, diabetes, chronic fatigue, fibromyalgia, anxiety, depression, eczema, and other chronic health problems.
You will be surprised to know that human gastrointestinal microbiota is a complex ecosystem of approximately 300 to 500 bacterial species, comprising nearly 2 million genes (the microbiome). Indeed, the number of bacteria within the gut is approximately 10 times that of all of the cells in the human body. At birth, the entire intestinal tract is sterile; the infant’s gut is first colonized by maternal and environmental bacteria during birth and continues to be populated through feeding and other contacts. Factors known to influence colonization include gestational age, mode of delivery (vaginal birth vs assisted delivery), diet (breast milk vs formula), level of sanitation, and exposure to antibiotics. by the age of 2.5 years, the
microbiota fully resembles the microbiota of an adult in terms of composition.
In humans, the composition of the flora is influenced not only by age but also by diet and socioeconomic conditions. In a study published in 2012 in “Nature” (high indexed journal) the interaction of diet and age was demonstrated, firstly, by a close relationship between diet and microbiota composition in the subjects and, secondly, by interactions between diet, the microbiota, and health status. It also concluded that non-digestible or undigested components (Fibre) of the diet may contribute substantially to bacterial metabolism; for example, much of the increase in stool volume resulting from the ingestion of dietary fibre is based on an augmentation of bacterial mass. Most recently, qualitative changes in the microbiota have been invoked in the pathogenesis of a global epidemic: obesity. It has been postulated that a shift in the composition of the flora toward a population dominated by bacteria that are more avid extractors of absorbable nutrients, which are then available for assimilation by the host could play a major role in obesity. Also there are enough evidence to support the hypothesis that the endogenous intestinal microflora plays a crucial role in the pathogenesis of Inflammatory bowel diseases and its variants and related disorders. Most Western populations over-consume highly refined, omnivorous diets of poor nutritional quality. Those diets are energy dense, high in animal protein, total and saturated fats, and
simple sugars but low in fruits, vegetables and other plant-based foods. Consequently, they are typically low in dietary fibre, non starch polysaccharides in general and resistant starch in particular. Cross-sectional studies have shown some evidence that Western-style diets are
associated with gut microbial populations that are typified by a Bacteroides enterotype (bad gut bacteria) whereas traditional diets rich in plant polysaccharides are associated with a Prevotella enterotype (good gut bacteria). Obesity is associated with an increased fecal Bacteroidetes:Firmicutes ratio relative to lean subjects. Replacing a habitual Western diet with one high in fiber elicited rapid (within 24 h) and marked alterations in fecal microbiota composition, although the changes were insufficient to produce a broad switch
from Bacteroides to Prevotella enterotype.  One mechanism by which fiber promotes and maintains bowel health is through increasing
digesta mass. Incompletely fermented fiber (e.g., insoluble non starch polysaccharides such as cellulose), increases digesta mass primarily through its physical presence and ability to adsorb water. An increase in digesta mass dilutes toxins, reduces intracolonic pressure, 
shortens transit time and increases defecation frequency. Fibers can also increase fecal mass to a lesser degree by stimulating fermentation, which leads to bacterial proliferation and increased biomass. Prebiotics are dietary substrates that selectively promote proliferation and/or activity of “beneficial” bacteria indigenous to the colon. The concept, first published by Gibson and Roberfroid in 1995, has been refined and redefined on several occasions. Prebiotics are defined currently as “selectively fermented ingredients that result in specific changes, in the
composition and/or activity in the GI microbiota, thus conferring benefit(s) upon host health”.
Dr Abhishek katakwar, Bariatric & amp; Metabolic surgeon from Asian Institute of Gastroenterology quote “Your body is a Temple. You are what you eat. Do not eat processed food, junk foods, filth, or disease carrying food, animals, or rodents. Some people say of these foods, ‘well, it tastes good;. Most of the foods today that statically cause sickness, 
cancer, and disease all taste good;s well seasoned and prepared poison. This is why so many people are sick; mentally, emotionally, physically, and spiritually; because of being hooked to  poison, instead of being hooked on the truth and to real foods that heal and provide you with good health and wellness.
1517037778
News article by Dr Abhishek Katakwar published in Deccan Chronicle (Date 21 january 2018) Our complete gastrointestinal tract is lined with microbes collectively called the microbiome, which includes bacteria, fungi, and even viruses. Though it sounds weird and even unhealthy, gut bacteria perform many important functions in the body, including aiding the immune system, producing the feel-good brain chemical serotonin, making energy available to the body from the food we eat, and disposing of foreign substances and toxins, though we always have a mixture of good and bad bacteria, sometimes the bad guys get the upper hand, causing an imbalance in gut bacteria, and can cause severe health problems than just stomach pain, gas, bloating, or diarrhea. Because 60-80% of our immune system is located in our gut, gut imbalances have been linked to obesity, hormonal imbalances, autoimmune diseases, diabetes, chronic fatigue, fibromyalgia, anxiety, depression, eczema, and other chronic health problems. You will be surprised to know that human gastrointestinal microbiota is a complex ecosystem of approximately 300 to 500 bacterial species, comprising nearly 2 million genes (the microbiome). Indeed, the number of bacteria within the gut is approximately 10 times that of all of the cells in the human body. At birth, the entire intestinal tract is sterile; the infant’s gut is first colonized by maternal and environmental bacteria during birth and continues to be populated through feeding and other contacts. Factors known to influence colonization include gestational age, mode of delivery (vaginal birth vs assisted delivery), diet (breast milk vs formula), level of sanitation, and exposure to antibiotics. by the age of 2.5 years, the microbiota fully resembles the microbiota of an adult in terms of composition. In humans, the composition of the flora is influenced not only by age but also by diet and socioeconomic conditions. In a study published in 2012 in “Nature” (high indexed journal) the interaction of diet and age was demonstrated, firstly, by a close relationship between diet and microbiota composition in the subjects and, secondly, by interactions between diet, the microbiota, and health status. It also concluded that non-digestible or undigested components (Fibre) of the diet may contribute substantially to bacterial metabolism; for example, much of the increase in stool volume resulting from the ingestion of dietary fibre is based on an augmentation of bacterial mass. Most recently, qualitative changes in the microbiota have been invoked in the pathogenesis of a global epidemic: obesity. It has been postulated that a shift in the composition of the flora toward a population dominated by bacteria that are more avid extractors of absorbable nutrients, which are then available for assimilation by the host could play a major role in obesity. Also there are enough evidence to support the hypothesis that the endogenous intestinal microflora plays a crucial role in the pathogenesis of Inflammatory bowel diseases and its variants and related disorders. Most Western populations over-consume highly refined, omnivorous diets of poor nutritional quality. Those diets are energy dense, high in animal protein, total and saturated fats, and simple sugars but low in fruits, vegetables and other plant-based foods. Consequently, they are typically low in dietary fibre, non starch polysaccharides in general and resistant starch in particular. Cross-sectional studies have shown some evidence that Western-style diets are associated with gut microbial populations that are typified by a Bacteroides enterotype (bad gut bacteria) whereas traditional diets rich in plant polysaccharides are associated with a Prevotella enterotype (good gut bacteria). Obesity is associated with an increased fecal Bacteroidetes:Firmicutes ratio relative to lean subjects. Replacing a habitual Western diet with one high in fiber elicited rapid (within 24 h) and marked alterations in fecal microbiota composition, although the changes were insufficient to produce a broad switch from Bacteroides to Prevotella enterotype.  One mechanism by which fiber promotes and maintains bowel health is through increasing digesta mass. Incompletely fermented fiber (e.g., insoluble non starch polysaccharides such as cellulose), increases digesta mass primarily through its physical presence and ability to adsorb water. An increase in digesta mass dilutes toxins, reduces intracolonic pressure, shortens transit time and increases defecation frequency. Fibers can also increase fecal mass to a lesser degree by stimulating fermentation, which leads to bacterial proliferation and increased biomass. Prebiotics are dietary substrates that selectively promote proliferation and/or activity of “beneficial” bacteria indigenous to the colon. The concept, first published by Gibson and Roberfroid in 1995, has been refined and redefined on several occasions. Prebiotics are defined currently as “selectively fermented ingredients that result in specific changes, in the composition and/or activity in the GI microbiota, thus conferring benefit(s) upon host health”. Dr Abhishek katakwar, Bariatric & amp; Metabolic surgeon from Asian Institute of Gastroenterology quote “Your body is a Temple. You are what you eat. Do not eat processed food, junk foods, filth, or disease carrying food, animals, or rodents. Some people say of these foods, ‘well, it tastes good;. Most of the foods today that statically cause sickness, cancer, and disease all taste good;s well seasoned and prepared poison. This is why so many people are sick; mentally, emotionally, physically, and spiritually; because of being hooked to poison, instead of being hooked on the truth and to real foods that heal and provide you with good health and wellness.
Crash diets may harm your heart

Cardiologist Isadore Rosenfeld, MD, a professor of clinical medicine at Weill Cornell Medical College, in New York City, and author of the forthcoming
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Crash diets may harm your heart Cardiologist Isadore Rosenfeld, MD, a professor of clinical medicine at Weill Cornell Medical College, in New York City, and author of the forthcoming "Doctor of the Heart: A Life in Medicine, " opposes crash diets (less than 1, 200 calories a day) and detox plans like the Master Cleanse. The Master Cleanse involves consuming a mixture of water, lemon juice, maple syrup, and cayenne pepper -- and nothing else -- for several days. Research suggests rapid weight loss can slow your metabolism, leading to future weight gain, and deprive your body of essential nutrients. What's more, crash diets can weaken your immune system and increase your risk of dehydration, heart palpitations, and cardiac stress. If you're overweight, slimming down is critical for your overall health. Even moderate weight loss can lower your risk of developing heart disease, diabetes, and some types of cancer. Bariatric surgery may be beneficial for selected patients. you may reach for best surgeon in Hyderabad, India. It's important to lose weight safely, which usually means slowly: Most experts recommend dropping just 1 to 2 pounds a week. And despite what some brand-name diets claim, the best way to do so is to exercise regularly and stick to a diet that limits saturated fat and sugars and emphasizes fruits and vegetables, lean meats and fish, and whole grains.
The Keto Diet Is Gaining Popularity, but Is It Safe?

The “keto” diet is any extremely low- or no-carbohydrate diet that forces the body into a state of ketosis. Ketosis occurs when people eat a low- or no-carb diet and molecules called ketones build up in their bloodstream.

Low carbohydrate levels cause blood sugar levels to drop and the body begins breaking down fat to use as energy.

1. Ketosis is actually a mild form of ketoacidosis. Ketoacidosis mostly affects people with type 1 diabetes. In fact, it is the leading cause of death of people with diabetes who are under 24 years of age.
2. Once your body enters ketosis, you also begin to lose muscle, become extremely fatigued, and eventually enter starvation mode. Then it actually becomes even harder to lose weight.
3. Keto diets should only be used under clinical supervision and only for brief periods.
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The Keto Diet Is Gaining Popularity, but Is It Safe? The “keto” diet is any extremely low- or no-carbohydrate diet that forces the body into a state of ketosis. Ketosis occurs when people eat a low- or no-carb diet and molecules called ketones build up in their bloodstream. Low carbohydrate levels cause blood sugar levels to drop and the body begins breaking down fat to use as energy. 1. Ketosis is actually a mild form of ketoacidosis. Ketoacidosis mostly affects people with type 1 diabetes. In fact, it is the leading cause of death of people with diabetes who are under 24 years of age. 2. Once your body enters ketosis, you also begin to lose muscle, become extremely fatigued, and eventually enter starvation mode. Then it actually becomes even harder to lose weight. 3. Keto diets should only be used under clinical supervision and only for brief periods.
Do you find it difficult to maintain weight loss after loosing 10 to 15 kg after different diet plans ????Hunger hormone ghrelin which tricks the bodies into thinking that it needs to eat more, may be the culprit. most people with obesity are able to lose weight, even on their own, but only 20 per cent manage to maintain the new lower weight. The studies shows that when we lose weight, the stomach releases greater amounts of the ghrelin hormone, which makes us feel hungry.
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Do you find it difficult to maintain weight loss after loosing 10 to 15 kg after different diet plans ????Hunger hormone ghrelin which tricks the bodies into thinking that it needs to eat more, may be the culprit. most people with obesity are able to lose weight, even on their own, but only 20 per cent manage to maintain the new lower weight. The studies shows that when we lose weight, the stomach releases greater amounts of the ghrelin hormone, which makes us feel hungry. "Everyone has this hormone, but if you've been overweight and then lose weight, the hormone level increases, " The best bariatric surgeon of town said. However, the level of ghrelin does not adjust over time, but remains high. This means it's likely that people who have been overweight will have to deal with increased hunger pangs for the rest of their lives, Dr Abhishek katakwar said. "People can lose motivation and have trouble following the diet and exercise advice. All of this makes it difficult to maintain the new lower weight” "Obesity is a daily struggle for the rest of one's life. We have to stop treating it as a short-term illness by giving patients some support and help, and then just letting them fend for themselves."
Who needs Bariatric & Metabolic Surgery: If your Efforts to lose weight with diet and exercise have been unsuccessful. Your body mass index (BMI) is 40 or higher. Your BMI is 35 or more and you have a serious weight-related health problem, such as type 2 diabetes, high blood pressure or severe sleep apnea.
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Who needs Bariatric & Metabolic Surgery: If your Efforts to lose weight with diet and exercise have been unsuccessful. Your body mass index (BMI) is 40 or higher. Your BMI is 35 or more and you have a serious weight-related health problem, such as type 2 diabetes, high blood pressure or severe sleep apnea.

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17.422594 78.457865 Bariatric (Obesity) And Metabolic (Diabetes) Surgery Bariatric and Metabolic surgery unit, Asian Institute of Gastroenterology, Kapadia lane, somajiguda, pin-500082, Telangana, INDIA
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